Alexander et al., Cancer Res 2004
(Cell Transformation, Neoplastic) :
In addition, these results suggest mechanisms by which normal cells may block Wnt signaling, inhibit insulin-like growth factor activity, and promote host recognition of neighboring tumor cells
Wang et al., J Bone Miner Res 2010
:
The results show that IGF-1 and the IGF- receptor (IGF1R) stimulate Wnt-4 expression and beta-catenin activation in growth plate chondrocytes