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MAPK6 — PLG

Text-mined interactions from Literome

Jingjing et al., Angiogenesis 2001 : CM from 293-VEGF183 cells treated with heparin or plasmin induced about a twofold increase in cell numbers and stimulated MAPK phosphorylation in HUVECs as compared with CM from untreated 293-VEGF183 cells or from heparin- or plasmin treated 293 cells containing the vector alone
Simkhovich et al., Cardiovasc Pathol 2003 (Myocardial Ischemia) : Protective genes included mitogen activated protein kinase activated protein kinase 3 ( MAPKAPK 3 ), heat shock proteins 70, 27, 22, B-crystalline, vascular endothelial growth factor, inducible nitric oxide synthase and plasminogen activator inhibitors 1 and 2
Ulfhammer et al., Journal of thrombosis and haemostasis : JTH 2006 (Cardiovascular Diseases...) : TNF-alpha mediated suppression of tissue type plasminogen activator expression in vascular endothelial cells is NF-kappaB- and p38 MAPK dependent
Zhang et al., J Immunol 2007 : Interaction of plasmin with annexin A2 resulted in the stimulation of ERK1/2 and p38 MAPK , cyclooxygenase-2, and PGE ( 2 ), leading to increased MMP-1 production
Zare et al., J Leukoc Biol 2008 : In contrast, Plg production did not require NF-kappaB, was only partly down-regulated by p38 MAPK inhibitor, and was efficiently inhibited by insulin, indicating a different mechanism for its induction
Tang et al., J Biol Chem 1998 : The urokinase-type plasminogen activator receptor mediates tyrosine phosphorylation of focal adhesion proteins and activation of mitogen activated protein kinase in cultured endothelial cells