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AKT1 — CD2AP

Text-mined interactions from Literome

Huber et al., Mol Cell Biol 2003 : One such target is Bad ; its phosphorylation and inactivation by 14-3-3 protects podocytes against detachment induced cell death, suggesting that the nephrin-CD2AP mediated AKT activity can regulate complex biological programs
Schiffer et al., J Biol Chem 2004 : CD2AP was required for early activation of anti-apoptotic phosphatidylinositol 3-kinase (PI3K)/AKT and extracellular signal regulated kinase 1/2 by TGF-beta ... CD2AP was not required for PI3K/AKT activation by insulin and epidermal growth factor, indicating that CD2AP is a selective mediator of anti-apoptotic TGF-beta signaling