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SOAT1 — STAT5A

Text-mined interactions from Literome

Smithgall et al., Oncogene 2000 : Together these approaches provide strong evidence that Stat activation , particularly of Stat3 and Stat5 , plays an important role in myeloid differentiation and survival
Vidal et al., Eur J Immunol 2001 : Furthermore, STAT1 activation but weak or no activation of STAT3 or STAT5 respectively, could be detected in MB-stimulated MDM. Using MonoMac6 cells, we observed high C/EBPbeta and AP-1 but weaker and transient NF-kappaB activation by MB.Moreover, the truncated form of C/EBPbeta, known to repress HIV-1 transcription, was detected in extracts from MB-treated THP-1 cells
Morcinek et al., Oncogene 2002 : We also found a new mechanism of activation of STAT5 by receptor tyrosine kinases, whereby direct interaction of the receptor kinase domain with the STAT protein in a phosphotyrosine independent way led to activation of STAT5 in terms of DNA binding and target gene expression
Yamaura et al., Am J Physiol Heart Circ Physiol 2003 (Myocardial Infarction...) : STAT signaling in ischemic heart : a role of STAT5A in ischemic preconditioning
Parafioriti et al., J Orthop Sci 2009 (Achondroplasia) : Chondrocytes in cartilage biopsies of ACH children were characterized by the presence of growth arrest mediated by STAT activation ( both STAT1 and STAT5 ) and increased expression of p21 and cyclin D1, whereas no expression of either p53 or cyclin D3 could be detected