Gene interactions and pathways from curated databases and text-mining

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MYLIP — NOTCH1

Text-mined interactions from Literome

Li et al., Cancer Res 2009 (Brain Neoplasms...) : Transfection of miR-34a down-regulated c-Met in human glioma and medulloblastoma cells and Notch-1 , Notch-2, and CDK6 protein expressions in glioma cells ... Transfection of miR-34a down-regulated c-Met in human glioma and medulloblastoma cells and Notch-1, Notch-2 , and CDK6 protein expressions in glioma cells ... miR-34a expression inhibited c-Met reporter activities in glioma and medulloblastoma cells and Notch-1 and Notch-2 3'-untranslated region reporter activities in glioma cells and stem cells
Guessous et al., Cell cycle (Georgetown, Tex.) 2010 (Brain Neoplasms...) : We showed that c-Met and Notch partially mediate the inhibitory effects of miR-34a on cell proliferation and cell death
Bao et al., Cancer Lett 2011 (Pancreatic Neoplasms) : Here we also report, for the first time, that over-expression of Notch-1 leads to increased expression of miR-21 , and decreased expression of miR-200b, miR-200c, let-7a, let-7b, and let-7c
Li et al., J Exp Med 2011 (Precursor T-Cell Lymphoblastic Leukemia-Lymphoma) : miR-451 but not miR-709 is conserved in humans, and human T-ALLs with activating NOTCH1 mutations have decreased miR-451 and increased MYC levels compared with T-ALLs with wild-type NOTCH1
Sureban et al., Journal of nanobiotechnology 2011 (Carcinoma...) : Lastly, DAPT mediated inhibition of Notch-1 resulted in HCT116 tumor growth arrest and down regulation of Notch-1 via a miR-144 dependent mechanism
Capuano et al., PloS one 2011 (Celiac Disease) : We found that high miR-449a levels targeted and reduced both NOTCH1 and KLF4 in HEK-293 cells
Nicoli et al., Dev Cell 2012 : miR-221 knockdown also prevented `` hyper-angiogenesis '' defects associated with Notch deficiency and miR-221 expression was inhibited by Notch signaling
Bae et al., Hum Mol Genet 2012 (Osteoporosis...) : Furthermore, miR-34c mediated post-transcriptional regulation of Notch signaling in osteoblasts is one possible mechanism to modulate the proliferative effect of Notch in the committed osteoblast progenitors which may be important in the pathogenesis of osteosarcomas
Kashat et al., American journal of translational research 2012 : We found that over-expression of miR-34a led to reduced expression of AR, PSA and Notch-1
Huang et al., BioMed research international 2013 : Since Notch signaling could inhibit muscle differentiation, a process in contrast with the effect of miR-1, miR-1 mediated repression of Notch signaling may contribute to the observed effects of miR-1 in mesenchymal stem cells
Jiang et al., Hypertension 2013 : In human endothelial cells, loss of miR-30a increased DLL4 protein levels, activated Notch signaling as indicated in Notch reporter assays, and augmented Notch downstream effector, HEY2 and EFNB2 ( ephrin-B2 ), expression