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HDAC1 — IL1A

Text-mined interactions from Literome

Yoshikawa et al., Arch Otolaryngol Head Neck Surg 2006 (Cholesteatoma, Middle Ear) : The messenger RNA expressions of LARC ( liver and activation regulated chemokine ), GMCSF ( granulocyte-macrophage colony stimulating factor ), epiregulin, ICAM1 ( intercellular adhesion molecule 1 ), and TGFA ( transforming growth factor alpha ) were more strongly up-regulated by IL-1alpha and/or IL-1beta in MECF than in SF, suggesting that these fibroblasts derived from different tissues retained their typical gene expression profiles
Enya et al., J Leukoc Biol 2008 (Melanoma) : These results indicate that the reduction in the activity and interaction of HDAC1 with Sp1 are critical for the constitutive IL-1 alpha gene expression
Pietrella et al., Infect Immun 2010 (Inflammation) : In particular Sap1, Sap2 , and Sap6 significantly induced interleukin-1ß (IL-1ß) , tumor necrosis factor alpha ( TNF-a ), and IL-6 production
Wessels et al., Immunobiology 2013 : The effects of histone deacetylase (HDAC)-inhibition by trichostatin A (TSA) on IL-1ß and TNFa expression and their promoter structures were measured in promyeloid HL-60 cells
Sarlo et al., Cell Immunol 1985 : The mouse macrophage line P388D1, also could bind SAP and display enhanced IL-1 production in response to SAP
Ku et al., Cytokine 1993 (Carcinoma, Hepatocellular...) : By contrast, gene expression of the major APR of the mouse, serum amyloid P-component (SAP) , a structural homologue of CRP, increased in response to either IL-1 or IL-6 under the same conditions