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FOS — GNRH1

Text-mined interactions from Literome

Mulvaney et al., J Biol Chem 1999 : These observations suggest that calcium influx through L-type channels is required for GnRH induced activation of ERK and c-Fos and that the influence of calcium lies downstream of protein kinase C
Roelle et al., J Biol Chem 2003 (MAP Kinase Signaling System) : Activation of c-Jun N-terminal kinase and p38 MAPK by GnRH was unaffected by EGFR or gelatinase inhibition that, however, suppressed GnRH induction of c-Jun and c-Fos
Fraley et al., Horm Behav 2004 : Central injections of GALP stimulate the secretion of gonadotropin releasing hormone (GnRH) and luteinizing hormone ( LH ) and induce the expression of Fos in several brain areas known to regulate male sexual behavior in the rat
Clarke et al., Endocrinology 2005 : Effects of changing gonadotropin releasing hormone pulse frequency and estrogen treatment on levels of estradiol receptor-alpha and induction of Fos and phosphorylated cyclic adenosine monophosphate response element binding protein in pituitary gonadotropes : studies in hypothalamo-pituitary disconnected ewes
Liu et al., Mol Endocrinol 2005 : Adenoviral expression of a kinase-inactive, dominant negative version of PKCdelta impaired GnRH activation of ERK, but not induction of c-Fos and LHbeta proteins, indicating that the novel PKCs signal to the ERK cascade
Coss et al., Mol Endocrinol 2007 : Inhibition of p38 by either of two different inhibitors or a dominant negative p38 kinase abrogates synergy on FSHbeta expression, reduces c-Fos induction by GnRH , and prevents the further increase in c-Fos levels that occurs with cotreatment
Blake et al., Brain Res 2010 : In naive-VNX animals, GnRH increased Fos in MeAv and MePv ... When the animals were sexually experienced before VNX, a condition in which GnRH does not enhance mating, i.c.v.-GnRH combined with MOB stimulation suppressed Fos expression in MePd
Ely et al., Mol Endocrinol 2011 : Although c-Fos is induced by a variety of stimuli in other cell types, in the gonadotropes, only GnRH induces c-Fos and through it FSHß ... GnRH induction of c-Fos in LßT2 cells requires the serum response factor (SRF) binding site, but not the Ets/ELK1 site
Binder et al., Mol Endocrinol 2012 (Calcium Signaling) : Additionally, although small interfering RNA specific for Nfat4 only marginally reduced GnRH regulation of Jun, Fos , and Atf3 mRNA accumulation, activity of an activator protein-1-responsive reporter construct was reduced by 48 %
Reddy et al., Mol Endocrinol 2013 : Here, we determined that GnRH induces rapidly both Egr1 and c-Fos , but specifically decreases the rate of c-Fos degradation
Glidewell-Kenney et al., Mol Endocrinol 2013 : We further show that this repression of GnRH transcription may involve enhanced c-Fos protein binding at novel activator protein-1 (AP-1) half-sites identified in enhancer 1 and the promoter, as well as chromatin remodeling at the promoter of the GnRH gene
Bruder et al., Mol Endocrinol 1996 : Phorbol ester inhibition of rat gonadotropin releasing hormone promoter activity : role of Fos and Jun in the repression of transcription