◀ Back to CXCL14
CXCL14 — EGF
Text-mined interactions from Literome
Azenshtein et al., Cancer Lett 2005
(Breast Neoplasms...) :
EGF potently
up-regulated CXCL8 secretion by breast tumor cells, and its effect was promoted by a consecutive treatment of the cells by estrogen and progesterone
Haim et al., Neoplasia (New York, N.Y.) 2011
(Breast Neoplasms) :
We found that stimulation by
EGF alone
induced the release of
CXCL8 through signaling pathways involving ErbB2, ErbB1, Erk, and phosphoinositide 3-kinase (PI3K)
Shaykhiev et al., Am J Respir Cell Mol Biol 2013
(Adenocarcinoma...) :
In vitro experiments revealed that the expression of CXCL14 is induced in the differentiated airway epithelium by cigarette smoke extract, and that
epidermal growth factor mediates
CXCL14 up-regulation in the airway epithelium through its effects on the basal stem/progenitor cell population
Hata et al., ISRN otolaryngology 2012
:
The
EGF effect on the expression of
CXCL14/BRAK was attenuated by the copresence of inhibitors of the EGF receptor, MEK, and ERK
Son et al., Journal of inflammation (London, England) 2013
:
Finally, CCL20 and
CXCL8 responded synergistically in
response to
EGF and TNF in OVCAR-3 and SKOV-3 cells