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CA2 — GRM1

Text-mined interactions from Literome

Francesconi et al., J Neurosci Res 2004 : We show that both mGluR1 and mGluR2 are activated by Ca2+ ( o ) in the absence of glutamate in the extracellular media
Rae et al., Neuropharmacology 2004 : Both mGluR1 and mGluR5 mediate Ca2+ release and inward currents in hippocampal CA1 pyramidal neurons
Dale et al., Methods Mol Biol 2011 (Calcium Signaling) : Specifically, we will describe a confocal imaging approach to simultaneously measure oscillilations in intracellular Ca2+ levels and PKC translocation to the plasma membrane in response to mGluR1 stimulation in transiently transfected human embryonic kidney ( HEK293 ) cells
Gabellini et al., Neuroreport 1993 : Glutamate activation of PI hydrolysis was reduced by pertussis toxin when mediated via mGluR1 alpha, while mGluR1T required the presence of extracellular Ca2+
Kawabata et al., J Biol Chem 1998 : Stimulation of mGluR1alpha induced an increase in [ Ca2+ ] i that consisted of an initial transient peak with a subsequent steady plateau or an oscillatory increase in [ Ca2+ ] i
Takechi et al., Nature 1998 (Calcium Signaling) : Our results indicate that these synaptic Ca2+ transients are mediated by activation of metabotropic glutamate-responsive mGluR1-type receptors and require inositol-1,4,5-trisphosphate mediated Ca2+ release from intradendritic stores