◀ Back to ABL1

ABL1 — CSF2

Text-mined interactions from Literome

Li et al., Blood 2001 (Cell Transformation, Neoplastic...) : Interleukin 3 and granulocyte-macrophage colony stimulating factor are not required for induction of chronic myeloid leukemia-like myeloproliferative disease in mice by BCR/ABL
Perrotti et al., Nat Genet 2002 (Blast Crisis...) : Here we show that expression of BCR-ABL in myeloid precursor cells leads to transcriptional suppression of the granulocyte colony stimulating factor receptor G-CSF-R ( encoded by CSF3R ), possibly through down-modulation of C/EBPalpha-the principal regulator of granulocytic differentiation
Leis et al., Leukemia & lymphoma 2004 (Blast Crisis...) : CSF levels were substantially below the concentration required for inhibition of BCR-ABL and killing of cell lines in vitro
Matsuguchi et al., J Biol Chem 1994 (Leukemia, Megakaryoblastic, Acute) : Shc phosphorylation in myeloid cells is regulated by granulocyte macrophage colony stimulating factor , interleukin-3, and steel factor and is constitutively increased by p210BCR/ABL
Zhang et al., Blood 1998 (Cell Transformation, Neoplastic...) : Bcr-Abl efficiently induces a myeloproliferative disease and production of excess interleukin-3 and granulocyte-macrophage colony stimulating factor in mice : a novel model for chronic myelogenous leukemia