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EGF — STK38
Text-mined interactions from Literome
Tartare et al., J Biol Chem 1991
:
These results suggest that
insulin-EGF chimeric receptor activation
stimulates at least one
serine/threonine kinase , which in turn phosphorylates the kinase-deficient EGF receptor
Cao et al., Science signaling 2009
:
The precise mechanism whereby
epidermal growth factor (EGF) activates the
serine-threonine kinase Akt and the mammalian target of rapamycin (mTOR) complex 1 ( mTORC1 ) remains elusive
Ahn et al., J Biol Chem 1990
:
Several separable
EGF stimulated
serine/threonine kinase activities were characterized in the preceding paper ( Ahn, N. G., Weiel, J. E., Chan, C. P., and Krebs, E.G. ( 1990 ) J. Biol. Chem. 265, 11487-11494 )
Saxena et al., Antioxid Redox Signal 2013
(Colonic Neoplasms...) :
Inhibition of AR also prevented the
epidermal growth factor induced phosphorylation of phosphatidylinositol 3-kinase (PI3K),
serine/threonine kinase ( AKT ), c-Jun, c-Fos, PTEN, and FOXO3a, and deoxyribonucleic acid ( DNA ) -binding activity of AP-1
King et al., J Biol Chem 1986
:
It has been suggested that
EGF could indirectly
activate a
protein-serine/threonine kinase , protein kinase C, that can phosphorylate the EGF receptor at threonine 654