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CA2 — NCS1

Text-mined interactions from Literome

Pan et al., J Neurosci 2002 : When cells were stimulated with histamine, NCS-1 overexpression led to higher exocytosis, as well as [Ca2+ ] i elevation
Koizumi et al., J Biol Chem 2002 : NCS-1 enhanced the UTP evoked exocytosis but not the depolarization evoked Ca2+ responses or exocytosis, suggesting that the enhancement by NCS-1 should involve phospholipase C-linked receptor mediated signals rather than the Ca2+ channels or exocytotic machinery per se. Taken together, NCS-1 enhances phosphoinositide turnover, resulting in enhancement of Ca2+ signaling and exocytosis
Winks et al., J Neurosci 2005 : The effects of bradykinin require additional components of action, which might involve IP3 induced Ca2+ release and consequent M-channel inhibition ( as proposed previously ) and stimulation of PIP2 synthesis by Ca2+ dependent activation of NCS-1
Hui et al., Development 2007 (Calcium Signaling) : We describe a model in which NCS-1 regulates cytosolic Ca2+ around the optimal window level to differentially control neurite extension and branching