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IL8 — ITGAM

Text-mined interactions from Literome

McClenahan et al., Am J Vet Res 2000 : Only IL-8 caused an increase in expression of CD11b by neutrophils
Furuta et al., J Leukoc Biol 2000 : Finally, co-incubation of post-hypoxic epithelia with human PMN induced IL-8 dependent expression of the PMN beta2-integrin CD11b/18
Manitz et al., Mol Cell Biol 2003 : Loss of S100A9 ( MRP14 ) results in reduced interleukin-8 induced CD11b surface expression, a polarized microfilament system, and diminished responsiveness to chemoattractants in vitro
Brickson et al., J Appl Physiol 2003 (Tendon Injuries) : The purpose of this study was to determine the role of the CD11b dependent respiratory burst in neutrophil oxidant generation and activation, interleukin-8 (IL-8) production, and myofiber damage after muscle stretch injury by using the monoclonal antibody M1/70 to block this pathway
Assis et al., Acta Haematol 2005 (Anemia, Sickle Cell) : RANTES ( 1-100 ng/ml ) had no significant effect on either normal or SCA neutrophil adhesion to FN. Flow-cytometric analyses demonstrated that IL-8 ( 10 ng/ml ) significantly augments CD11b ( Mac-1 integrin subunit ) expression on SCA neutrophils, but not normal neutrophils
Nandate et al., Anesth Analg 2005 : We also analyzed the effect of NAC on interleukin-8 (IL-8) induced expression of CD11b in human whole blood
Detmers et al., J Immunol 1991 : NAP-2, melanoma growth stimulatory activity, and two forms of NAP-1/IL-8 ( ser-NAP-1/IL-8 and ala-NAP-1/IL-8, consisting of 72 and 77 amino acids, respectively ), each caused an increase in the expression of CD11b/CD18 ( CR3 ) and CR1, which was accompanied by a decrease in the expression of leukocyte adhesion molecule-1 ( LAM-1, LECAM-1 )
Ozaki et al., J Trauma 2007 : Stimulation with IL-8 significantly upregulated CD11b expression ( 5.3 +/- 1.7 to 7.5 +/- 2.7, p < 0.01 ) and this IL-8 induced upregulation of CD11b was also inhibited by ethanol pretreatment ( p < 0.001 )
Fuxman Bass et al., Mol Immunol 2008 : Granulocyte-macrophage colony stimulating factor increased IL-8 and IL-1beta secretion, and CD11b-upregulation induced by single stranded bacterial DNA
Detmers et al., J Exp Med 1990 : NAP-1/IL-8 promoted additional adhesive interactions between CD11b/CD18 and the biosynthetic precursor of LPS, lipid IVa, fibrinogen, and endothelial cells, suggesting that NAP-1/IL-8 may promote leukocyte adhesion in vivo that could lead to recruitment of PMN to sites of tissue inflammation
Lundahl et al., Scand J Immunol 2012 (Blister) : IL-8 from local subcutaneous wounds regulates CD11b activation
Au et al., J Immunol 1994 : Zymosan induced IL-8 release from human neutrophils involves activation via the CD11b/CD18 receptor and endogenous platelet activating factor as an autocrine modulator
Molad et al., Biochem J 1994 : In contrast, similar to previous studies, recombinant C5a, recombinant IL-8 and FMLP all stimulated increased expression of CD11b/CD18 ( 170-260 % of basal, P < 0.001, n = 5 ) and shedding of L-selectin ( 56-75 % reduction from basal, P < 0.001, n = 5 ) at similar concentrations and with similar potencies ( EC50 = 2, 5, and 3 nM respectively )
Lundahl et al., Scand J Immunol 1996 : The quantitative levels of CD11b/CD18 , but not CD29 and CD61, was increased by fMLP, but not RANTES nor IL-8
Lee et al., Immunol Lett 1996 : Thus the suppression of CD11b/CD18 expression by tepoxalin may involve IL-8
Knowles et al., Am J Physiol 1997 (Anoxia) : During reoxygenation, both whole blood PMN phagocytosis and bactericidal activity were reduced in the presence of IL-8 , TNF-alpha, or IL-1beta, and in subsets of PMN with reduced phagocytosis H/R reduced CD64, CD32w, CD16, CD35, and CD11b/CD18 expression in the presence of each cytokine
Goodman et al., Arch Surg 1998 (Respiratory Distress Syndrome, Adult...) : Effect of early, fulminant ARDS and normal plasma on spontaneous apoptosis, CD16, and CD11-b expression in PMNs in vitro ; levels of IL-8 in plasma ; correlation of extracellular IL-8 concentration with rate of PMN apoptosis ; and effect of IL-8 blockade on PMN apoptosis, CD16, and CD11-b expression in ARDS and normal plasma