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MMP9 — TCF3

Text-mined interactions from Literome

Troussard et al., Oncogene 2000 (Glioblastoma) : The integrin linked kinase (ILK) induces an invasive phenotype via AP-1 transcription factor dependent upregulation of matrix metalloproteinase 9 (MMP-9)
Simon et al., Exp Cell Res 2001 (Neoplasm Invasiveness...) : Considering that TPA is a potent activator of AP-1, we hypothesized that this transcription factor might also be required for p38 pathway dependent MMP-9 regulation
Kato et al., J Biol Chem 2005 (Melanoma, Experimental) : A transcription factor ( s ) other than NF kappa B may also be involved in the MMP-9 expression
Ray et al., J Immunol 2005 (Arthritis) : Using progressively deleted and mutant promoter constructs of MMP-9, we show that serum amyloid A-activating factor ( SAF)-1, a novel transcription factor , and the AP-1 family of proteins cooperatively regulate cytokine mediated induction of MMP-9 in the resident cells of the joint capsule
Rajapakse et al., Bioorg Med Chem 2007 (Fibrosarcoma) : Further, transfection of promoter genes of MMPs and their transcription factors clearly exhibited that inhibition of MMP-2 and MMP-9 was due to down-regulation of transcription factor , NF-kappaB
Tsai et al., Mol Cell Biol 2008 : Our previous studies indicated that coactivator and transcription factor phosphorylation may allow for rapid regulation of MMP-9 expression
Balasubramanian et al., J Biol Chem 2011 : The NF-?B transcription factor is required for full induction of MMP-9 by TNF-a
Yen et al., J Biol Chem 2011 (MAP Kinase Signaling System) : PKA, PI3K, and ERK inhibitors abolished PGE2- and cAMP induced c-Fos and MMP-9 up-regulation, and ERK activation was required for the binding of activator protein 1 (AP-1) transcription factor to the MMP-9 promoter
Ganguly et al., J Biol Chem 2013 : To reveal the role of AP-1 transcription factor in MMP-9 gene expression, we have used reporter gene constructs with specifically mutated AP-1 gene promoter sites
Tyagi et al., J Mol Cell Cardiol 1997 : Results suggested that the increase in MMP promoter activity by heparin is due to a specific transcription factor binding to MMP-1 promoter sequence