Gene interactions and pathways from curated databases and text-mining

◀ Back to CCR1

CCL2 — CCR1

Pathways - manually collected, often from reviews:

  • KEGG Chemokine signaling pathway: CCL1/CCL11/CCL13/CCL14/CCL15/CCL16/CCL17/CCL18/CCL19/CCL2/CCL20/CCL21/CCL22/CCL23/CCL24/CCL25/CCL26/CCL27/CCL28/CCL3/CCL3L1/CCL3L3/CCL4/CCL4L1/CCL4L2/CCL5/CCL7/CCL8/CX3CL1/CXCL1/CXCL10/CXCL11/CXCL12/CXCL13/CXCL14/CXCL16/CXCL2/CXCL3/CXCL5/CXCL6/CXCL9/IL8/PF4/PF4V1/PPBP/XCL1/XCL2 → CCR1/CCR10/CCR2/CCR3/CCR4/CCR5/CCR6/CCR7/CCR8/CCR9/CX3CR1/CXCR1/CXCR2/CXCR3/CXCR4/CXCR5/CXCR6/XCR1 (protein-protein, activation)

Protein-Protein interactions - manually collected from original source literature:

Studies that report less than 10 interactions are marked with *

Text-mined interactions from Literome

Kim et al., Biochem Pharmacol 2004 : The intracellular Ca ( 2+ ) concentration and the chemotactic activity increased in 9-CRA treated cells in response to the CCR1 dependent chemokines Lkn-1, MIP-1alpha, and RANTES, and the CCR2-specific chemokine MCP-1
Wang et al., Zhongguo Shi Yan Xue Ye Xue Za Zhi 2006 (Leukemia, Myelogenous, Chronic, BCR-ABL Positive) : It is concluded that P210 ( bcr/abl ) fusion protein inhibits the expression of MIP-1alpha and CCR-1 in chronic myeloid leukemia cells, but does not inhibit MCP-1 and CCR-2 mRNA expressions in these leukemia cells