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BCR — SOAT1
Text-mined interactions from Literome
Su et al., J Biol Chem 1999
:
Finally, we show that co-ligation of CD19 with the BCR results in increased tyrosine phosphorylation of STAT1 relative to BCR cross linking alone, establishing CD19 as a positive modulator of
BCR mediated
STAT activation
Wang et al., Oncogene 2007
:
We found that activated
BCR results in tyrosine phosphorylation of
JAK-STAT , which required Lyn
Frank et al., Leukemia 1996
(Cell Transformation, Neoplastic) :
BCR/abl leads to the constitutive activation of
Stat proteins, and shares an epitope with tyrosine phosphorylated Stats
Ahmed et al., Oncogene 1998
:
Our results indicate that although both constitutively activated cEpoR and BCR-ABL oncogenes induce growth-factor independence in Ba/F3 cells, only
BCR-ABL is able to protect cells from etoposide and serum-deprivation induced apoptosis and
induce a strong constitutive activation of
STAT factors, suggesting a role for these molecules in the anti-apoptotic activity of BCR-ABL