◀ Back to SYT1
POLDIP2 — SYT1
Text-mined interactions from Literome
Ha et al., Proc Natl Acad Sci U S A 2004
:
Here, we demonstrate an additional mechanism whereby a significant reduction of proinflammatory gene expression such as IL-1beta, tumor necrosis factor alpha, and inducible nitricoxide synthase in PARP-1 ( -/- ) glial cells is linked to defective inflammatory stimuli induced
p38MAPK mediated phosphorylation of ATF-2 and cAMP-response element binding protein and phosphorylation of NF-kappaB
p65
Peirce et al., PloS one 2010
:
Moreover, LPS induced activation of the MAP kinases ERK and
p38 was
enhanced in cells over expressing Themis2 whereas the activation of JNK, IRF3 or NF-kappaB
p65 , was unaffected