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UCSC Genome Browser Gene Interaction Graph
Gene interactions and pathways from curated databases and text-mining

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POLDIP2 — SYT1

Text-mined interactions from Literome

Ha et al., Proc Natl Acad Sci U S A 2004 : Here, we demonstrate an additional mechanism whereby a significant reduction of proinflammatory gene expression such as IL-1beta, tumor necrosis factor alpha, and inducible nitricoxide synthase in PARP-1 ( -/- ) glial cells is linked to defective inflammatory stimuli induced p38MAPK mediated phosphorylation of ATF-2 and cAMP-response element binding protein and phosphorylation of NF-kappaB p65
Peirce et al., PloS one 2010 : Moreover, LPS induced activation of the MAP kinases ERK and p38 was enhanced in cells over expressing Themis2 whereas the activation of JNK, IRF3 or NF-kappaB p65 , was unaffected