◀ Back to VIP

NGF — VIP

Text-mined interactions from Literome

Hill et al., Peptides 2002 : VIP treatment also induced an increase of NGF-like immunoreactivity (NGF-IR) within the neural tube preparation tissue
Thippeswamy et al., J Mol Neurosci 2007 : Nitric oxide-NGF mediated PPTA/SP, ADNP, and VIP expression in the peripheral nervous system
Korkmaz et al., J Mol Neurosci 2010 (Parkinsonian Disorders) : Therefore, the protective effect of VIP on motor function is possibly related to the increased levels of GABA in the VATh, and its neuroprotective actions may be mediated by the release of NGF from brain mast cells
Missale et al., Proc Natl Acad Sci U S A 1996 : Activation of the dopamine D2 receptor subtype, which physiologically controls prolactin release, resulted in a complete inhibition of vasoactive intestinal peptide stimulated NGF secretion in vitro, whereas the specific D2 antagonist ( - ) -sulpiride stimulated NGF secretion in vivo, suggesting that the anterior pituitary is a possible source of circulating NGF