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BCL10 — IRF6

Text-mined interactions from Literome

Fukui et al., J Immunol 2003 : LPS stimulation induced the expression of Fas, caspase 8, cellular FLIP Bfl-1/A1, and Bcl-x , but not FasL, TNFR p55, Bak, Bax, and Bad at the transcriptional level
Liu et al., J Biol Chem 2004 : In response to lipopolysaccharide (LPS) stimulation, BCL10 was recruited to TLR4 signaling complexes and associated with Pellino2, an essential component down-stream of BCL10 in the TLR4 pathway ... In response to lipopolysaccharide (LPS) stimulation, BCL10 was recruited to TLR4 signaling complexes and associated with Pellino2, an essential component down-stream of BCL10 in the TLR4 pathway
Sano et al., Res Vet Sci 2005 : By real-time quantitative PCR analysis, it was indicated that Bcl-xL and Bax levels in canine neutrophils were significantly affected by LPS stimulation
Yu et al., FASEB J 2005 (Inflammatory Bowel Diseases) : Western blot showed that LPS in high glucose increased the levels of anti-apoptotic Bcl-2 and Bcl-X ( L, ) and did not change proapoptotic Bax
Ramana et al., Free Radic Biol Med 2007 : In addition, AR inhibition prevented the LPS induced down-regulation of Bcl-xl and up-regulation of Bax and Bak in macrophages
Bhattacharyya et al., Am J Physiol Gastrointest Liver Physiol 2007 (Inflammation) : Bcl10 mediates LPS induced activation of NF-kappaB and IL-8 in human intestinal epithelial cells
Lee et al., J Pineal Res 2009 (Body Weight...) : In western blot analysis, LPS reduced the levels of Bcl-XL and procaspase-3 in SD mice