Gene interactions and pathways from curated databases and text-mining

◀ Back to TNF

B2M — TNF

Text-mined interactions from Literome

Zheng et al., J Immunol 2000 : Transduced Bcl-2 did not reduce TNF mediated NF-kappaB activation or constitutive expression of class I MHC molecules
Steck et al., Zentralbl Gynakol 2002 : The suppressive effect of HLA-G and HLA-E on the secretion of TNF-alpha ( Th1 cytokine ), IL-10 ( Th2 cytokine ) and IL-8 ( chemokine ) by immature DC could be interpreted as further evidence for the central immunotolerance role of HLA-G and HLA-E during early pregnancy
Ntrivalas et al., J Soc Gynecol Investig 2006 : The expression of human leukocyte antigen (HLA)-G on trophoblast cell lines did not affect CD56+/TNF-alpha ( + ) NK cell levels under these experimental conditions
Doukas et al., J Immunol 1990 : TNF but not IL-1 also increases class I MHC Ag expression
Hoon et al., Cancer Res 1991 (Melanoma) : IL4, IFN, or TNF alone enhanced human leukocyte antigen class I, DR, and beta 2-microglobulin antigen expression
Schiller et al., Cancer Res 1991 (Neoplasms) : TNF significantly ( P less than 0.05 for all ) enhanced serum beta 2-microglobulin , serum neopterin, and serum interleukin-2 receptor ( Tac antigen ) levels
Leszczynski et al., Am J Pathol 1990 : Finally, TNF alpha induced class I MHC expression was inhibited strongly by IL-1 alpha
Zhao et al., Hum Immunol 2013 : Functional analysis revealed that ß2m-free HLA-G promoted xenogeneic cytotoxic lysis of BRIN-BD11 cells by natural killer ( NK ) cells and increased production of IL-1ß, TNF-a , and IFN-?
Wedgwood et al., Cell Immunol 1988 : TNF in combination with IFN-gamma enhances HUVEC expression of HLA-alpha, B2m , and HLA-DQ greater than that observed with either mediator alone
Zuber et al., Int J Cancer 1988 (Glioma) : The surface expression of HLA-ABC ( class I ) antigens and beta 2-microglobulin , was significantly enhanced by TNF-alpha alone on every cell line and clone tested
Stephens et al., J Autoimmun 1997 (Diabetes Mellitus, Type 1) : In the current study we have examined the effect of the same inhibitor of NF-kappaB on class I major histocompatibility complex ( MHC ) protein expression in NIT-1 cells and shown that inhibition of NF-kappaB activation decreased basal and TNF induced class I MHC levels