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Kimura et al., Scand J Immunol 2000 (Lymphoma, B-Cell) : In the absence of serum or with the addition of anti-CD14 monoclonal antibodies, however, LPS-stimulation induced neither the up-regulation of CD14 and TGF-beta mRNA nor an increase in IgA secretion
Bone et al., Int Immunol 2001 : Nuclear NF-kappaB is induced in B cells by engagement of either the BCR or CD40 or by stimulation with lipopolysaccharide (LPS)
Hindle et al., Infect Immun 2002 (Salmonella Infections...) : Six of nine volunteers elicited anti-serovar Typhi lipopolysaccharide (LPS) immunoglobulin A (IgA) antibody secreting cell (ASC) responses , with three of three vaccinees receiving 10 ( 8 ) and two of three receiving 10 ( 9 ) CFU which elicited high-titer LPS-specific serum IgG
Takatsu et al., Nihon rinsho. Japanese journal of clinical medicine 2004 (Agammaglobulinemia...) : Functional analysis of the IL-5Ralpha-/- and IL-5-/- mice revealed that IL-5 plays critical roles in the homeostatic proliferation, cell survival and activation of mature B-1 cells and regulates IgA production in the mucosal tissues in response to LPS
Park et al., Mol Cells 2005 : Mechanisms underlying the effects of LPS and activation induced cytidine deaminase on IgA isotype expression ... We explored the effect of LPS on the expression of AID during B cell differentiation, and the role of AID in IgA isotype expression ... TGF-b1 and AID were required for IgA expression, and LPS contributed to TGFb1 induced IgA production largely by inducing AID ... Interestingly, LPS repressed AID transcription in sIgA+ B cells but still stimulated IgA production mainly by increasing the rate of IgA secretion
Kirkpatrick et al., J Infect Dis 2005 (Typhoid Fever) : Immunogenicity data included Salmonella Typhi lipopolysaccharide (LPS)-specific immunoglobulin (Ig) A antibody secreting cells ( enzyme linked immunospot [ ELISPOT ] assay ), IgG serologic responses to Salmonella Typhi LPS , lymphocyte proliferation, and interferon (IFN)- gamma production
Eckmann et al., Immunology 1992 (Lymphoma, B-Cell) : IL-5 as well as LPS significantly increases IgA secretion of 4F10 cells, whereas TGF beta 1, a cytokine known to stimulate isotype switching to IgA among surface IgM bearing B cells, inhibits IgA secretion
Shockett et al., J Immunol 1991 (Lymphoma, B-Cell) : We also present evidence that IL-4 may provide other signals, distinct from transcriptional targeting, that increase LPS induced switching to IgA
Bretschneider et al., Vet Immunol Immunopathol 2007 (Escherichia coli Infections) : The serum and fecal IgG and IgA antibody responses of adult cattle to E. coli O157 : H7 intimin, translocated intimin receptor ( Tir ), E. coli secreted proteins ( Esp ) A, EspB and O157 lipopolysaccharide (LPS) in response to infection were determined
Hashizume et al., Infect Immun 2008 : Interestingly, Salmonella lipopolysaccharide (LPS)-specific fecal IgA responses were not elicited in PP-null mice, while serum IgG anti-LPS antibodies were identical to those of control mice
Maes et al., Neuro Endocrinol Lett 2008 (Fatigue Syndrome, Chronic) : Subchronic intake of those NAIOSs significantly attenuates the initially increased IgA and IgM responses to LPS of gram negative bacteria ... A good clinical response is significantly predicted by attenuated IgA and IgM responses to LPS , the younger age of the patients, and a shorter duration of illness ( < 5 years )
Shamsuzzaman et al., Vaccine 2009 : Plasma IgA and IgG responses to CTB and IgA responses to both O1 and O139 LPS were elevated
Khan et al., Scand J Infect Dis 2010 (Cholera...) : Ogawa infection produced higher plasma vibriocidal as well as IgG responses to cholera toxin B subunit, toxin coregulated pilus subunit and lipopolysaccharide (LPS) ; higher IgA responses to LPS in ` antibody in lymphocyte supernatant ' ( ALS ) specimens were also seen
Cavallo et al., Clin Exp Immunol 1991 (Autoimmune Diseases...) : Our results indicate that : ( i ) LPS induces IgA deficiency in normal mice concurrently with PBA ; ( ii ) IgA deficiency is a feature of lupus-prone mice ; ( iii ) LPS accentuates naturally occurring PBA and IgA deficiency in lupus-prone mice ; and ( iv ) LPS induced, or LPS enhanced, IgA deficiency and PBA in normal and lupus-prone mice persist long after withdrawal of LPS ... Because immune defects are common in autoimmune diseases and IgA deficiency is prevalent in patients with systemic lupus erythematosus, we investigated : ( i ) whether LPS might induce IgA deficiency in normal mice ; ( ii ) whether IgA deficiency might be a feature in lupus-prone mice ; ( iii ) whether, if present in lupus-prone mice, IgA deficiency could be further accentuated by LPS ; and ( iv ) whether the effects of LPS on IgA concentrations of normal and lupus-prone mice might be reversible upon withdrawal of LPS
Shipley et al., Comp Med 2010 : Animals receiving 10 ( 11 ) cfu S. dysenteriae 1 intragastrically consistently developed signs of shigellosis characterized by the onset of diarrhea and dysentery within 2 to 3 d. Administration of as many as 10 ( 9 ) cfu S. dysenteriae 1 intraduodenally did not elicit signs characteristic of infection in macaques despite fecal shedding of bacteria for as long as 10 d. S. dysenteriae 1 administered intraduodenally at 10 ( 9 ) cfu or intragastrically at 10 ( 11 ) cfu elicited robust IgG and IgA antibody responses to LPS
Sjöblom-Hallén et al., Mucosal Immunol 2010 : Moreover, IgG1 and IgA production induced by LPS and IL-10 were enhanced by the addition of CT to cultures of PP or splenic B cells
Alam et al., Clinical and vaccine immunology : CVI 2011 (Cholera) : Individuals with natural cholera developed prominent vibriocidal and plasma anti-cholera toxin B subunit ( CtxB ) and lipopolysaccharide (LPS) IgG and IgA responses , but these responses returned to baseline by 1 year of follow-up
Feller et al., Vaccine 2011 : We compared IgA and IgG responses to serotype-specific Shigella LPS using ELISPOT and ALS in subjects following vaccination or infection with Shigella
Tarique et al., Clinical and vaccine immunology : CVI 2012 (Cholera...) : The LPS-specific IgA responses in stool were induced by transcutaneous ( P < 0.01 ) but not subcutaneous immunization
Leung et al., Clinical and vaccine immunology : CVI 2012 (Cholera) : In contrast, children recovering from natural cholera infection developed prominent LPS IgG and IgA MBC responses , as well as CtxB IgG MBC responses
Maes et al., Acta Psychiatr Scand 2013 (Inflammation) : The IgA responses to LPS were correlated with lysozyme
Kim et al., Immune network 2012 : Moreover, 100 nM CpG treatment abrogated either LPS induced IgM or LPS/TGF-ß1 induced IgA and IgG2b production, although B cell growth was enhanced by CpG under the same culture conditions ... Further, 10 nM CpG, unlike 100 nM, neither abrogated the LPS/TGF-ß1-nor the LPS/RA induced IgA production
Johnson et al., Clinical and vaccine immunology : CVI 2012 (Cholera) : Plasma IgG, IgM, and IgA responses to Ogawa OSP and LPS in Ogawa infected patients also correlated well with each other ( R = 0.60, 0.60, and 0.92, respectively ; P < 0.01 ) ... Mucosal IgA immune responses to OSP and LPS were also similar
Chen et al., Cell Immunol 1990 : TGF-beta 1 is a potent costimulator of LPS induced IgA responses
Kawaguchi et al., Clin Immunol Immunopathol 1988 (Glomerulonephritis, IGA) : These results suggest that the persistent use of LPS directly suppresses gut associated lymphoreticular tissue ( GALT ), and that the lower IgA response in GALT induced by LPS causes suppression of glomerular IgA deposition in ddY mice
Alberini et al., Eur J Immunol 1987 (Lymphoma) : Cells from the monoclonal B cell lymphoma I.29 expressing surface IgM ( mu + ) are capable of differentiating in vitro to IgM secretion and of switching to IgA or IgE production in response to lipopolysaccharide (LPS) stimulation
Cryz et al., J Lab Clin Med 1988 : Immunization evoked a vigorous IgA1 and IgA2 antibody response to LPS , which was long lived
Luzi et al., Clin Exp Immunol 1986 (Dysgammaglobulinemia...) : Decreased IgA responses to both LPS and PWM were consistently observed in cultures of MNC from all of the nine children with IgA deficiency
Németh et al., Acta Microbiol Hung 1985 (Arthritis, Rheumatoid) : PBMs from both groups could be induced by LPS to secrete IgM and IgA rheumatoid factors ( RF ), antinuclear and anti-beta-2-microglobulin autoantibodies
Layton et al., Int Arch Allergy Appl Immunol 1983 : LPS stimulation resulted in an increase in total IgA ( ninefold ), IgM ( sixfold ) and IgG ( eightfold ) synthesis over unstimulated cultures and similar increases were seen in IgA anti-TNP ( eightfold ), IgM anti-TNP ( fivefold ) and IgG anti-TNP ( twelvefold )
Islam et al., Infect Immun 1995 (Dysentery, Bacillary) : The anti-LPS IgA2 response was more prominent in SDIP than in SFIP
Fukuba et al., Immunobiology 1994 : The reactivity of thymic B cells to IL-5 is found to be significantly lower than that of splenic B cells both in proliferative responses and LPS induced IgM and IgA antibody responses
Thomson et al., J Rheumatol 1994 (Arthritis, Reactive...) : There was a prolonged salivary IgA anti-LPS response in both the ReA/ReE and DYS ( dysentery alone ) patients compared with unexposed controls
Shockett et al., J Immunol 1993 : In studies aimed at identifying other signaling pathways that promote switching, we discovered that inhibitors of the nuclear enzyme poly ( ADP-ribose ) polymerase ( PARP ) increase LPS induced switching to IgA in the B cell lymphoma 1.29 mu and to IgG1 in LPS + IL-4 treated splenic B cells
Kärnell et al., Vaccine 1993 (Dysentery, Bacillary) : Challenge infection of vaccinated monkeys elicited serum IgA and IgG responses to the homologous S. flexneri 2a LPS in three monkeys each ( 0.005 < or = p < or = 0.025 )
Forrest et al., Vaccine 1993 : Following the primary PBL anti-LPS IgA response to an oral course of live vaccine in all subjects ( 6/6 ), boosting with a single oral dose of live vaccine resulted in 0/6 responders, while 2/11 subjects responded after a single dose of parenteral vaccine
Brownlie et al., Microb Pathog 1993 : A specific IgA response to LPS was detected after immunization with the OMP containing liposomes but not with the LPS containing liposomes, indicating adjuvant activity of the proteins
Harriman et al., J Clin Invest 1996 (IgA Deficiency) : Notably, I alpha exon-deficient B cells did not require TGF-beta for IgA class switch since stimulation with LPS alone led to IgA expression
Cohen et al., Clin Diagn Lab Immunol 1996 (Dysentery, Bacillary) : Seventy-four percent of the volunteers with a significant IgA or IgG anti-LPS response in serum at convalescence or 14 days after vaccination showed a similar response in urine
Söderhäll et al., J Clin Invest 1997 (Cystitis...) : In fact, the globoside binding strain DS17, but not the mutant DS17-1, unable to bind to membrane bound globoside, elicited a secretory IgA response to LPS in urine
Yan et al., Toxicology 1997 : Consistent with IL-5 's previously documented role in IgA production, anti-IL-5 decreased IgA levels to background in cultures of both control and VT-exposed PP or spleen cells in the presence of either PMA + ION or LPS
Ishizaka et al., Int J Immunopharmacol 1998 : Neither FOM nor TGF-beta inhibited LPS induced IgA antibody responses, whereas dexamethasone ( DX ) reduced polyclonal IgM, IgG and IgA antibody responses wholly