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CISH — TNF

Text-mined interactions from Literome

Colson et al., Endocrinology 2000 : Although IL-1/beta and TNFalpha alone induced only weakly the expression of SOCS-3 and CIS , these cytokines strongly potentiated the induction of these two SOCS by GH ... Considering the ability of these SOCS to inhibit the JAK-STAT pathway induced by GH, these results suggest that the overexpression of SOCS-3 and CIS mRNAs induced by IL-1beta and TNFalpha or by endotoxin in vivo may play a role in the GH resistance induced by sepsis
Kurimoto et al., J Invest Dermatol 1992 (Dermatitis, Contact) : Because the effects of UVB radiation and cis-UCA are reversed by anti-TNF alpha antibodies, we propose that UVB radiation impairs the induction of CH in mice by converting trans-UCA to cis-UCA within the epidermis ; cis-UCA in turn causes the local release of TNF alpha , which thwarts sensitization by its ability to alter the functional program of epidermal Langerhans cells, thereby preventing the induction of CH
Fasshauer et al., J Endocrinol 2004 (Insulin Resistance) : Recently, it was demonstrated that induction of suppressor of cytokine signaling (SOCS)-3 by TNFalpha and GH is an important mechanism by which these cytokines impair insulin sensitivity
Kurimoto et al., J Immunol 1992 (Dermatitis, Contact) : Based on these findings we propose that UVB radiation impairs the induction of contact hypersensitivity in mice by converting trans-urocanic acid to cis-UCA within the epidermis ; cis-UCA in turn causes the local release of TNF-alpha , which thwarts sensitization by its ability to trap epidermal Langerhans cells transiently within the epidermis, and thereby prevents the immunogenic signal from reaching the draining lymph node where activation of unprimed, Ag-specific T cells must occur
Xu et al., J Neuroimmunol 2006 (Inflammation) : The studies demonstrated that 9-cis-RA inhibited the production of nitric oxide ( NO ) as well as the pro-inflammatory cytokines TNF-alpha , IL-1beta and IL-12 p40 by LPS stimulated microglia ... In LPS stimulated astrocytes, 9-cis-RA inhibited NO and TNF-alpha production but had not effect on IL-1beta, IL-6 and MCP-1 secretion
Dawson et al., BMC immunology 2006 : Stimulation of human PBMCs and purified T cells with ATRA and 9-cis-RA increased mRNA and protein levels of IL-4, IL-5, and IL-13 and decreased levels of IFN-gamma, IL-2, IL-12p70 and TNF-alpha upon activation with anti-CD3 and/or anti-CD28 mAbs
Rigby et al., Oncogene 2007 (Colonic Neoplasms...) : In vitro, SOCS3 overexpression reduced proliferation, IL-6 mediated STAT3 activation and tumor necrosis factor (TNF) alpha mediated NF-kappaB activation
Pogrebniak et al., Surgery 1991 : Therefore, we hypothesized that cis-dichlorodiammine platinum (CDDP) , mitomycin-C, and doxorubicin hydrochloride ( Adriamycin ), by the release of ROS, would increase macrophage TNF production
Prêle et al., J Immunol 2008 : However, SOCS1 suppressed the sustained production of TNF-alpha by primary human monocytes and synovial fluid macrophages ex vivo
Carow et al., PLoS Pathog 2013 : Instead, SOCS3 expression in infected macrophages and DCs prevented the IL-6 mediated inhibition of TNF and IL-12 secretion and contributed to a timely CD4+ cell dependent IFN-? expression in vivo
Renner et al., Nutrients 2013 : Quantitative real time polymerase chain reaction analyses were performed to evaluate the expression of interleukin (IL)-4, IL-10, interferon ( IFN ) -?, tumor necrosis factor (TNF)-a and peroxisome proliferator activated receptor ( PPAR ) -? in response to cis-9,trans-11 and LA
Hughes et al., Biochem Biophys Res Commun 1995 : We present evidence that both the cis- and trans- forms of RA and to a lesser extent, the RA precursor beta-carotene, can inhibit recombinant human TNF cytolytic activity in mouse L-929 cells
Moodycliffe et al., Immunology 1994 (Dermatitis, Contact) : Thus cis-UCA does not act through TNF-alpha induction or by influencing DC migration, and other studies indicate that histamine-like receptors in the skin may be involved