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CCL5 — TNF

Pathways - manually collected, often from reviews:

• WikiPathways EBV LMP1 signaling: NFKB1/RELA → Complex of CXCL8-IFNB1-CCL20-TNF-CCL5 (activation)
  

Text-mined interactions from Literome

Mustafa et al., Eur J Oral Sci 2001 : The expression of Rantes/CCL5-mRNA and protein production, induced by TNFalpha , was evident at 6 h and thereafter increased continuously during the study period ( 24 h )
Nanki et al., J Immunol 2001 (Arthritis, Rheumatoid) : On the other hand, CCL2, CCL5 , and CXCL12 were expressed on RA FLS, and their production was regulated by TNF-alpha , IL-1beta, and TGF-beta1
Kanda et al., Biochem Pharmacol 2006 : Suppressive effects of antimycotics on tumor necrosis factor-alpha induced CCL27, CCL2, and CCL5 production in human keratinocytes ... We examined in vitro effects of antimycotics on TNF-alpha induced CCL27, CCL2, and CCL5 production in human keratinocytes ... Antimycotics ketoconazole and terbinafine hydrochloride suppressed TNF-alpha induced CCL27, CCL2, and CCL5 secretion and mRNA expression in keratinocytes in parallel to the inhibition of NF-kappaB activity while fluconazole was ineffective ... Anti-prostaglandin E2 ( PGE2 ) antiserum or antisense oligonucleotides against PGE2 receptor EP2 or EP3 abrogated inhibitory effects of ketoconazole and terbinafine hydrochloride on TNF-alpha induced NF-kappaB activity and CCL27, CCL2, and CCL5 production, indicating the involvement of endogenous PGE2 in the inhibitory effects ... Carboxyheptyl imidazole also suppressed TNF-alpha induced NF-kappaB activity and CCL27, CCL2, and CCL5 production ... These results suggest that ketoconazole and terbinafine hydrochloride may suppress TNF-alpha induced NF-kappaB activity and CCL27, CCL2, and CCL5 production by increasing PGE2 release from keratinocytes
Lee et al., J Immunol 2008 : IL-17 strongly repressed TNF-alpha stimulated expression of CXCL10, CXCL11, and CCL5 , but synergized with TNF-alpha for induction of CXCL8, CXCL1, and CCL20 mRNAs
Machado et al., Microbes Infect 2008 : Interestingly, T. cruzi infected NOS2-deficient CMs produced significantly higher levels of CCL2, CCL4, CCL5 and CXL2 in the presence of IFN-gamma and TNF
Imaizumi et al., Immunol Lett 2009 (Arthritis, Rheumatoid) : In addition, knockdown of RIG-I partially inhibited the TNF-alpha induced expression of CC chemokine ligand (CCL) 5 , a chemokine with chemotactic activity toward lymphocytes and monocytes
Subileau et al., J Neuropathol Exp Neurol 2009 (Inflammation...) : Tumor necrosis factor strongly increased the production of CCL2, CCL5 , and CXCL8 ; interferon gamma upregulated CXCL10 exclusively
Xu et al., Stem Cells 2009 : C/EBPbeta knockdown also inhibited the synergistic expression of CXCL1, inducible nitric oxide synthase, and CCL5 in response to concomitant IFNgamma and TNFalpha
Homma et al., Int Arch Allergy Immunol 2010 : Cooperative activation of CCL5 expression by TLR3 and tumor necrosis factor-alpha or interferon-gamma through nuclear factor-kappaB or STAT-1 in airway epithelial cells
Tannheimer et al., Respir Res 2012 (Inflammation...) : Evaluation of roflumilast ( 1-10 µM ) showed no significant inhibition alone on TGFß1 induced ET-1 and CTGF mRNA transcripts, ET-1 and FN protein production, alpha smooth muscle expression, or TNF-a induced secretion of CXCL10, CCL5 and GM-CSF
Chen et al., Biochem Biophys Res Commun 2013 (Dermatitis, Atopic...) : LL-37 inhibited the increase of CXCL10 and CCL5 induced by TNF-a- and/or IFN-? but enhanced that of CXCL8