We have a suspicion that you are an automated web bot software, not a real user. To keep our site fast for other users, we have slowed down this page. The slowdown will gradually disappear. If you think this is a mistake, please contact us at genome-www@soe.ucsc.edu. Also note that all data for hgGeneGraph can be obtained through our public MySQL server and all our software source code is available and can be installed locally onto your own computer. If you are unsure how to use these resources, do not hesitate to contact us.
UCSC Genome Browser Gene Interaction Graph
Gene interactions and pathways from curated databases and text-mining

◀ Back to FAS

FAS — RNF19A

Text-mined interactions from Literome

Brenner et al., Cell Death Differ 1998 : Here, we demonstrate that Fas receptor triggered activation of the acidic sphingomyelinase, consumption of sphingomyelin, release of ceramide, and subsequent activation of JNK and p38-K are regulated by caspases
Ivanov et al., Oncogene 2000 (Melanoma) : Forced expression of a dominant negative form of p38 ( p38-ASP ) or treatment with p38 pharmacological inhibitor, SB203580, increases NF-kappaB activity, Fas expression and the levels of UVC induced apoptosis in late stage melanoma cells
Park et al., Mol Cancer Ther 2009 (Carcinoma, Renal Cell) : GST-MDA-7 killing was, in parallel, dependent on inactivation of extracellular signal regulated kinase 1/2 and on CD95 induced p38 mitogen activated protein kinase and c-jun NH ( 2 ) -terminal kinase-1/2 signaling
Juo et al., Mol Cell Biol 1997 : In this study Fas activated the stress-responsive mitogen activated protein kinases, p38 and JNK, within 2 h in Jurkat T lymphocytes but not the mitogen-responsive kinase ERK1 or pp70S6k