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LIF — STAT1

Text-mined interactions from Literome

Zhang et al., Exp Eye Res 2005 : Retinas from embryonic and neonatal stages showed that STAT3 but not STAT1 was activated in response to ciliary neurotrophic factor (CNTF), leukemia inhibitory factor (LIF) , fibroblast growth factor-1 (FGF1), fibroblast growth factor-2 (FGF2), epidermal growth factor (EGF), interferon-alpha ( IFN-alpha ) and interferon-gamma (IFN-gamma) in distinct patterns
Dekanty et al., J Biol Chem 2006 : LIF and OSM specifically trigger STAT1 cytoplasmic to nuclear translocation, whereas PGF2alpha fails to do so
Kurdi et al., Int J Biochem Cell Biol 2012 (MAP Kinase Signaling System) : Unexpectedly, LIF induced STAT1 activation was unaffected by GSH depletion
Fahmi et al., Cell Signal 2013 (Hypertrophy...) : In the absence of sIL-6R, IL-6 did not induce STAT1/3 phosphorylation, whereas IL-6/sIL-6R and LIF induced STAT1 and STAT3 phosphorylation
Ray et al., J Clin Invest 1996 (Pituitary Neoplasms) : 1 nM LIF rapidly induced tyrosyl phosphorylation of STAT 1 and STAT 3 proteins, as well as tyrosyl phosphorylation of a 115-kD protein, coimmunoprecipitated with STAT 1