◀ Back to ATP5O

ATP5O — IL1A

Text-mined interactions from Literome

Tattersall et al., Cell Physiol Biochem 2005 : The upregulation of ATPase activity by IL-1 was inhibited by the NOS inhibitor L-NAME and by the NO scavenger PTIO
Chan et al., J Food Sci 2009 (Neurodegenerative Diseases) : H ( 2 ) O ( 2 ) or MPP ( + ) treatment significantly decreased Na ( + ) -K ( + ) -ATPase activity, elevated caspase-3 activity and levels of interleukin (IL)-1 , IL-6, and tumor necrosis factor (TNF)-alpha ( P < 0.05 ) ; and the pretreatments from these agents significantly restored Na ( + ) -K ( + ) -ATPase activity, suppressed caspase-3 activity and release of IL-1, IL-6, and TNF-alpha ( P < 0.05 )
Bulut et al., Ulus Travma Acil Cerrahi Derg 2011 (Pancreatitis) : While plasma amylase, lipase, IL-1ß , and TNF-a levels, and tissue MDA and MPO levels significantly increased in rats with cerulean induced AP, tissue GSH and Na+-K+-ATPase activity significantly reduced
Brisseau et al., J Biol Chem 1996 : Finally, stimulation of both cAMP dependent protein kinase and protein kinase C was required for the stimulatory effect of IL-1 on V-ATPase activity
McTiernan et al., Circ Res 1997 : The effects on transcript levels were gene specific ; IL-1 beta induced transcripts for inducible NO synthase (iNOS), did not alter GAPDH transcripts, and reduced sarcoplasmic reticulum Ca ( 2+ ) -ATPase ( 65 % of control, P < .001 ) transcripts