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IGF1 — PLCB1

Text-mined interactions from Literome

Martelli et al., J Cell Biochem 1999 : Insulin-like growth factor-I dependent stimulation of nuclear phospholipase C-beta1 activity in Swiss 3T3 cells requires an intact cytoskeleton and is paralleled by increased phosphorylation of the phospholipase
Xu et al., J Biol Chem 2001 : Protein kinase C alpha -mediated negative feedback regulation is responsible for the termination of insulin-like growth factor I-induced activation of nuclear phospholipase C beta1 in Swiss 3T3 cells
Xu et al., Mol Cell Biol 2001 : Stimulation of Swiss 3T3 cells with insulin-like growth factor I (IGF-I) caused rapid nuclear translocation of activated ERK and concurrently induced phosphorylation of nuclear PLC beta1 , which was completely blocked by the MEK inhibitor PD 98059