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IL1A — PDCD11

Text-mined interactions from Literome

Wu et al., Biochem Biophys Res Commun 2011 (Alzheimer Disease...) : LXA ( 4 ) down-regulated the protein expression of IL-1ß and TNFa, attenuated the gene expressions of IL-1ß and TNFa, inhibited the degradation of I?Ba, inhibited translocation of NF-?B p65 subunit into the nucleus induced by ß-amyloid in the cortex and hippocampus of mice, and in Aß-stimulated BV2 cells, and the inhibitory effects were dose dependently elevated
Rautava et al., J Pediatr Gastroenterol Nutr 2012 : Conditioning with TGF-ß2 inhibited IL-1ß induced I?B-a degradation and NF-?B p65 nuclear translocation, which may partially result from TGF-ß2 induced changes in the expression of genes in the NF-?B signaling pathway detected by cDNA microarray and qPCR
Wall et al., Oxidative medicine and cellular longevity 2013 : Luteolin and kaempferol decreased IL-1ß induced NF- ? B p65 DNA binding activity and nuclear c-Jun expression
Dunger et al., J Autoimmun 1996 : Since inhibition of NO production was always paralleled by a reduced ability of IL-1 to induce programmed cell death , this radical appears to be involved in this process