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GPI — IL6

Text-mined interactions from Literome

Kahlke et al., Cytokine 2000 (Hemorrhage...) : In contrast, IL-1beta and IL-6 production by Mbeta from young males was suppressed and IL-10 production enhanced following trauma-haemorrhage, whereas Mstraight phi from aged males produced elevated levels of IL-1beta and IL-6 and suppressed levels of IL-10 following trauma-haemorrhage
Kojima et al., Proc Natl Acad Sci U S A 2005 : Furthermore, the expression of the STAT3 ( 534-770 ) region in the nuclei of STAT3-knockdown cells enhanced the IL-6 induced NLK activation in a dose dependent manner but not the TGFbeta induced NLK activation
Yoshimura et al., Transplantation 1991 : Interestingly, however, AMF did not suppress interleukin 3 (IL-3) and interleukin 6 (IL-6) production, as well as IL-2 receptor expression
Lu et al., Journal of hematology & oncology 2008 (Multiple Myeloma) : It was also demonstrated that PHI inhibited IL-6 receptor expression and VEGF production in the RPMI8226 cells, and reactivated p21 expression
Conroy et al., PloS one 2009 (Inflammation...) : Plasmodium falciparum GPI ( pfGPI ) enhanced C5a receptor expression ( CD88 ) on monocytes, and the co-incubation of monocytes with C5a and pfGPI resulted in the synergistic induction of cytokines ( IL-6 , TNF, IL-1beta, and IL-10 ), chemokines ( IL-8, MCP-1, MIP1alpha, MIP1beta ) and the anti-angiogenic factor sFlt-1 in a time and dose dependent manner
von Kleist et al., Immunobiology 1990 : LPS, IL6 , and IL4 caused induction of up to 15 % Fo+ and Ia+ M phi after a 4 d culture period
Holladay et al., Prostaglandins Leukot Essent Fatty Acids 1993 : These data suggest that PGI2 ( but not TXA2 ) is involved in AA-induced IL-6 release from peritoneal M phi
Marcinkiewicz et al., Cell Immunol 1993 : In contrast, both PGE2 and PGI2 potentiated the release of IL-3 and IL-6 which both play an important role in stimulating haemopoesis after inflammation
Pruimboom et al., Prostaglandins Leukot Essent Fatty Acids 1994 : We conclude that LPS stimulation of hp-M phi from liver disease results in similar production of IL-1 beta, IL-6 and TNF-alpha, but that the profile of the eicosanoid production of these M phi stimulated with LPS and A23187 differs from M phi of other origin and species
Todd et al., J Matern Fetal Med 1996 : IL-1, but not IL-6 or IL-8, caused a dose- and time dependent increase in the production of both PGI2 and PGE2
Wen et al., Prostaglandins 1996 : Inhibitory effects of interleukin-6 on release of PGI2 by cultured human pulmonary artery smooth muscle cells
Mullol et al., Neuropeptides 1997 : After 4 h of incubation, 10 ( -7 ) M PHI also increased IL-6 release by 74 % ( P < 0.01 )
Sasaki et al., Blood 1997 (Hematologic Neoplasms...) : Phorbol 12-myristate 13-acetate ( PMA ), interleukin-1 (IL-1), IL-3, IL-6, granulocyte-macrophage colony stimulating factor ( GM-CSF ), thrombopoietin (TPO), and tumor necrosis factor-alpha (TNF-alpha) enhanced PGI2-R mRNA expression