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CD79B — JUN
Text-mined interactions from Literome
Perrotti et al., Mol Cell Biol 2000
:
FUS proteolysis is induced by
c-Jun , is
suppressed by
BCR-ABL or Jun kinase 1, and does not depend on c-Jun transactivation potential, ubiquitination, or its interaction with Jun kinase 1
Inabe et al., FEBS Lett 2002
(Agammaglobulinemia) :
Recent studies have shown that Btk plays an important role in
BCR mediated
c-Jun NH ( 2 ) -terminal kinase ( JNK ) 1
activation ; however, the mechanism by which Btk participates in the JNK1 response remains elusive
Mancini et al., Traffic 2009
(Leukemia, Myelogenous, Chronic, BCR-ABL Positive) :
In particular, constitutive tyrosine kinase ( TK ) activity of p210
BCR-ABL blocks
c-Jun N-terminal kinase (JNK) phosphorylation leading to 14-3-3 sigma phosphorylation at a critical residue ( Ser ( 186 ) ) for c-ABL binding in response to DNA damage