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AHSA1 — INS

Text-mined interactions from Literome

Gosmanov et al., Diabetes 2002 : PI 3-kinase inhibitors also abolished insulin stimulated p38 MAPK phosphorylation in the plantaris muscle and Akt phosphorylation in both muscles
Carlson et al., Diabetes 2003 (Diabetes Mellitus, Type 2...) : Insulin stimulation did not increase p38 phosphorylation in either healthy control subjects or type 2 diabetic patients
Schliess et al., J Biol Chem 2004 : Insulin increases [ Tyr ( P ) ( 418 ) ] Src, [ Tyr ( P ) ( 397 ) ] FAK, and dual p38 ( MAPK ) phosphorylation by about 2-fold
Thirone et al., Diabetes 2006 (Insulin Resistance) : Insulin dependent phosphorylation of IRS1/2 and Shc was not affected by siJAK2, but insulin induced phosphorylation of the mitogen activated protein kinases ( MAPKs ) extracellular signal related kinase, p38 , and Jun NH2-terminal kinase and their respective upstream kinases MKK1/2, MKK3/6, and MKK4/7 was significantly lowered when JAK2 was depleted, correlating with a significant drop in insulin mediated cell proliferation
Kummer et al., J Biol Chem 1997 : Insulin ( 50 ng/ml ) inhibited p38 activity by approximately 70 % and blocked apoptosis by at least 80 %