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Vosler et al., Proc Natl Acad Sci U S A 2011
(Ischemia) :
Finally, calpain inhibition following global ischemia in vivo blocked decreases in eIF4G1 , facilitated protein synthesis, and increased neuronal viability in ischemia-vulnerable hippocampal CA1 neurons
Neumar et al., J Cereb Blood Flow Metab 1998
(Ischemic Attack, Transient) :
Because eIF-4G is a substrate of calpain, these studies were undertaken to examine whether calpain I activation during global brain ischemia contributes to the degradation of eIF-4G in vivo