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FANCC — STAT1

Protein-Protein interactions - manually collected from original source literature:

Studies that report less than 10 interactions are marked with *

• IRef Biogrid Interaction: FANCC — STAT1 (physical association, affinity chromatography technology) Pang et al., Mol Cell Biol 2000*
• IRef Biogrid Interaction: FANCC — STAT1 (direct interaction, two hybrid) Reuter et al., Exp Cell Res 2003
• IRef Biogrid Interaction: FANCC — STAT1 (physical association, affinity chromatography technology) Pang et al., Blood 2001*
• IRef Biogrid Interaction: FANCC — STAT1 (direct interaction, pull down) Pang et al., Mol Cell Biol 2000*
• IRef Hprd Interaction: STAT1 — FANCC (in vitro) Pang et al., Mol Cell Biol 2000*
• IRef Hprd Interaction: STAT1 — FANCC (in vivo) Pang et al., Mol Cell Biol 2000*
• IRef Ophid Interaction: STAT1 — FANCC (aggregation, interologs mapping) Brown et al., Bioinformatics 2005
• IRef Ophid Interaction: STAT1 — FANCC (aggregation, confirmational text mining) Pang et al., Mol Cell Biol 2000*

Text-mined interactions from Literome

Pang et al., Blood 2001 (Fanconi Anemia) : FANCC is also required for optimal activation of STAT1 in response to cytokine and growth factors and for suppressing cytokine induced apoptosis by modulating the activity of double stranded RNA dependent protein kinase
Fagerlie et al., J Immunol 2004 (Fanconi Anemia) : Consequently, loss of a functional FANCC results in decreased activation of STAT1 following IFN-gamma stimulation