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ACHE — FAS

Text-mined interactions from Literome

Blasina et al., Cell Tissue Res 2000 : However, FAS , at concentrations in which changes of morphological parameters were observed, did not inhibit the AChE activity as measured histochemically
Quillfeldt et al., Braz J Med Biol Res 1991 : 1. We examined the effect, in rats, of an intraseptal microinjection of fasciculin (FAS) , an irreversible peptide acetylcholinesterase (AChE) inhibitor , on a ) AChE activity measured in septum and hippocampus, b ) 3H-quinuclidinyl benzylate ( 3H-QNB ) and 3H-oxotremorine ( 3H-OXO ) binding to hippocampal cholinergic muscarinic receptors, c ) 3H-flunitrazepam ( 3H-FNZ ) binding to hippocampal benzodiazepine receptors as a control for QNB and OXO binding, d ) acquisition and retention in three different behavioral paradigms, i.e., water finding ( in which there is concomitant habituation to the apparatus ), step-down inhibitory avoidance, and shuttle avoidance
Dajas et al., Brain Res 1993 : The effects of the peptide fasciculin (FAS) , a potent inhibitor of acetylcholinesterase (AChE) have been examined, following unilateral microinfusion, on tissue levels of monoamines in the rat substantia nigra and concomitant circling behaviour ... Although FAS inhibited 87 % of total AChE , the levels of dopamine and its metabolites remained unchanged
Marchot et al., J Biol Chem 1993 : All the specific binding of 125I-FAS3 to AChE is prevented by FAS3 as from D. angusticeps venom ( Kd = 0.4, 14, and 25 pM, respectively )