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BAX — CDK2

Text-mined interactions from Literome

Morris et al., J Neurosci 2001 : However, either p53 deficiency or the inhibition of CDK activity alone inhibited Bax translocation, cytochrome c release, and caspase-3-like activation
Choi et al., Apoptosis 2007 : Thus, we suggest that Cdk2 activity is involved in the mitochondrial translocation of Bax , which plays an important role in the mitochondrial membrane permeability transition during apoptotic progression
Wang et al., J Asian Nat Prod Res 2009 : 1 in high concentrations induced Bax , caspase-3, and cyclin dependent kinase (CDK) inhibitor P21 expression, whereas the expressions of cyclin CDK2 , Bcl-2, and proliferating cell nuclear antigen ( PCNA ) were decreased by 1 treatment
Gil-Gómez et al., EMBO J 1998 : We found that Cdk2 activation during thymocyte apoptosis can be regulated by p53, Bax and Bcl-2