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MAPK9 — RASA1
Text-mined interactions from Literome
Van Slyke et al., Mol Cell Biol 2005
(Breast Neoplasms) :
Dok-R 's ability to attenuate EGF-driven
MAPK activation is
independent of its ability to recruit
rasGAP , a known attenuator of MAPK activity, suggesting an alternate Dok-R mediated pathway
Zhao et al., Mol Cell Biol 2006
:
On the other hand, Dok-1 negatively
regulates PDGF induced
MAPK activation by acting on
Ras-GAP and at least one other Dok-1 interacting protein
Szczur et al., Blood 2006
(MAP Kinase Signaling System) :
We further suggest that CDC42GAP mediated extracellular signal regulated kinase ( ERK ) activity regulates motility associated with podosome-like structures at the cell leading edge, while
CDC42GAP induced p38 (
MAPK ) phosphorylation regulates directed migration by antagonizing filopodia assembly
Vilá de Muga et al., Oncogene 2009
:
In ER-negative MDA-MB-436 cells, overexpression of p120GAP, but not CAPRI or a
p120GAP mutant lacking the AnxA6 binding domain
inhibits Ras/MAPK activity
Yao et al., Oncogene 1995
:
GAP overexpression also
inhibited stimulation of
mitogen activated protein ( MAP ) kinase and induction of immediate-early genes in response to NGF