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CCL19 — EPHB2

Text-mined interactions from Literome

Kohout et al., J Biol Chem 2004 : The mechanism by which CCL19 activates ERK1/2 was determined to be beta-arrestin dependent, because it is reduced both by depletion of beta-arrestin-2 with small interfering RNA and by elimination of the phosphorylation sites in the tail of the receptor
Shannon et al., J Biol Chem 2012 : While CCL19 stimulated ERK5 ( flox/flox ) naïve T cells showed increased migration to EDG-1 ligands at 48 h, the migration of ERK5 ( flox/flox ) /Lck-Cre T cells remained at a basal level