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Toma et al., Biochem Biophys Res Commun 2009
(Hyperglycemia...) :
AGE-LDL stimulated
p22(phox) expression by activating p38 MAP kinase and NF-kB, and MCP-1 expression by activating NF-kB, as demonstrated by the use of specific inhibitors ( SB203580 and Bay11-7085 ) ... The addition of 25mM glucose in the culture medium enhanced the
effect of
AGE-LDL , but also of nLDL, on RAGE,
p22(phox) , NOX4, p67(phox), and MCP-1 gene expression
Zhao et al., Free radical research 2011
:
The present study demonstrated that oxidized
LDL ( oLDL )
increased the generation of superoxide and hydrogen peroxide ( H ( 2 ) O ( 2 ) ), the abundances of NADPH oxidase (NOX)4, NOX2,
p22-phox and lectin-like oLDL receptor-1 ( LOX-1 ) in wild-type or heat shock factor-1 (HSF1)-deficient mouse embryo fibroblasts (MEF)
Fu et al., Vascul Pharmacol 2013
:
Our results showed that in the
presence of
ox-LDL , XJP-1 significantly attenuated ox-LDL mediated cytotoxicity, apoptosis, caspase-3 activation, reactive oxygen species ( ROS ) generation, and NADPH oxidase subunit (
p22phox and p47phox ) expression in HUVECs