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PCNA — PDGFB

Text-mined interactions from Literome

Sharma et al., Am J Physiol 1999 : The eNOS transfer significantly inhibited ( P < 0.05 ) PDGF-BB induced proliferating cell nuclear antigen ( PCNA ) and cyclin A expression in VSM cells compared with cells transfected with the control vector
Sanz-González et al., Biochem Biophys Res Commun 2004 : STI571 abrogates PDGF-BB dependent cyclin D1 and cyclin A protein expression and inhibits transcriptional activation of reporter genes driven by the human cyclin A gene promoter ... We also found that STI571 abrogates PDGF-BB dependent activation of extracellular regulated kinase 1 and 2 ( ERK1/2 ), and forced activation of these factors impaired STI571 dependent inhibition of both cyclin A promoter activity and SMC proliferation
Pan et al., Biochem Cell Biol 2007 : Our results revealed that PDGF-BB significantly increased the expressions of TGF-beta1 and PCNA , and the increase in PCNA can be partially inhibited by neutralizing anti-TGF-beta1 antibody ... These findings suggest that PDGF-BB promotes PCNA expression in VSMCs partially through TGF-beta1 overexpression, and that the TGF-beta signalling system involves the molecular mechanism of PDGF-BB in VSMCs
Karpurapu et al., J Biol Chem 2008 (Carotid Stenosis) : Inhibition of nuclear factors of activated T cell ( NFAT ) activation by cyclosporin A ( CsA ) and VIVIT suppressed PDGF-BB induced cyclin A expression and CDK2 activity, resulting in blockade of VSMC in the G ( 1 ) phase
Xu et al., Clin Exp Pharmacol Physiol 2011 : In addition, TSG significantly inhibited PDGF-BB induced phosphorylation of Rb and the expression of cyclin D1, CDK4, cyclin E, CDK2 and PCNA
Liu et al., Mol Cell Biochem 2011 : Berberine and DHEAS decreased the expression of CDK2, CDK4, PCNA , cyclin D1, and cyclin E, which was induced by PDGF-BB