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CCL2 — IKBKB

Text-mined interactions from Literome

Goebeler et al., Blood 2001 : Consistent with that finding, stimulation with NiCl ( 2 ) or TNFalpha activated IkappaB kinase-beta (IKKbeta), and transient transfection of dominant negative IKKbeta strongly inhibited NiCl ( 2 ) - and TNFalpha induced MCP-1 expression
Gawaz et al., Thromb Haemost 2002 : Furthermore, overexpression of kinase-inactive IKK-beta KA, as well as NIKKA, inhibited platelet- or IL-1 beta induced kappa B-, MCP-1- or VCAM-1 dependent transcription
Wen et al., J Pharmacol Exp Ther 2006 : For the first time, we have demonstrated that in human fibroblast-like synoviocytes, TNFalpha- or interleukin (IL)-1beta induced monocyte chemoattractant protein-1 regulated on activation, normal T cell expressed and secreted and production is IKKbeta dependent
Catley et al., Mol Pharmacol 2006 (Pulmonary Disease, Chronic Obstructive) : As observed above, ICAM-1, IL-6, IL-8, GM-CSF, RANTES, MCP-1 , GROalpha, NAP-2, and ENA-78 expression was reduced by the IKK inhibitors
Röder-Stolinski et al., Inhal Toxicol 2008 : In the presence of IKK-NBD , a specific NF-kappa B inhibitor, or the inhibitors of the p38 MAP kinase SB 203580 and SB 202190, the chlorobenzene related MCP-1 release was suppressed, suggesting an involvement of both pathways in the chlorobenzene induced expression of MCP-1