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EPO — SOAT1

Text-mined interactions from Literome

Bittorf et al., Biol Chem 1999 (Leukemia, Erythroblastic, Acute) : In conclusion, SHP1 regulates the transcriptional activity stimulated by the EPO induced JAK/STAT and MAPK pathways and is involved in the signaling machinery responsible for erythroid differentiation and suppression of apoptosis
Lai et al., Oncogene 2005 (Carcinoma, Squamous Cell...) : Erythropoietin mediated activation of JAK-STAT signaling contributes to cellular invasion in head and neck squamous cell carcinoma
Chen et al., J Ethnopharmacol 2009 : The effects of TSPG-pretreatment on Epo induced JAK(2) and STAT ( 5 ) tyrosine phosphorylation were analyzed by immunoprecipitation ... TSPG markedly enhanced Epo induced tyrosine phosphorylation of JAK(2) and STAT ( 5 ) in UCB-MNCs
Kim et al., J Cell Biochem 2012 : Recently, we demonstrated that Epo activates JAK/STAT signaling in hematopoietic stem cells ( HSCs ), leading to the production of bone morphogenetic protein 2 (BMP2) and bone formation and that Epo also directly activates mesenchymal cells to form osteoblasts in vitro
Cokic et al., Journal of translational medicine 2012 : The goal of this study was to observe the function of EPO activation of JAK-STAT and PI3K/AKT pathways in the development of erythroid progenitors from hematopoietic CD34+ progenitor cells, as well as to distinguish early EPO target genes in human erythroid progenitors during ontogeny
Penta et al., J Biol Chem 1995 : This EPO dependent activation of STAT proteins was maximum within 10 min of exposure of the cells to 10 units of EPO/ml, the concentration of EPO required for maximum STAT activation