Gene interactions and pathways from curated databases and text-mining

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CCL24 — IL4

Text-mined interactions from Literome

Watanabe et al., J Immunol 2002 : Eotaxin-2 generation is differentially regulated by lipopolysaccharide and IL-4 in monocytes and macrophages ... Indeed, IL-4 suppressed the generation of eotaxin-2 by LPS stimulated monocytes ... Moreover, IL-4 , but not LPS, up-regulated the production of eotaxin-2 by macrophages
Meyer-Hoffert et al., Int Arch Allergy Immunol 2003 : IL-4 and to a lesser extent IL-13 and IFN-gamma stimulated the production of MCP-3 and -4, eotaxin and eotaxin-2 immunoreactivities
Lezcano-Meza et al., Allergy 2003 (Nasal Polyps) : IL-4 induced eotaxin-2/CCL24 in a dose dependent manner with concentrations as low as 0.1 ng/ml being able to induce eotaxin-2/CCL24
Kobayashi et al., Ann Allergy Asthma Immunol 2004 : Eotaxin-1 and eotaxin-3, but not eotaxin-2 , mRNA expressions were induced by stimulation with interleukin (IL) 13 or IL-4
Kagami et al., Clin Exp Immunol 2005 (Dermatitis, Atopic) : The production of CCL24 in HaCaT cells was slightly enhanced by IL-4 and that of CCL26 was strongly enhanced by IL-4 and IL-13
van Wetering et al., Mol Immunol 2007 : Epithelial differentiation is a determinant in the production of eotaxin-2 and -3 by bronchial epithelial cells in response to IL-4 and IL-13 ... TNFalpha reduced IL-4 induced eotaxin-2 release in submerged cultures but not in ALI-cultures ; no effects on eotaxin-3 synthesis were observed
Errahali et al., J Interferon Cytokine Res 2009 (Asthma) : Results demonstrate that CCL26-siRNA treatments decreased interleukin-4 induced CCL26 and CCL24 expression by > 70 %
Wu et al., Clin Exp Allergy 2009 (Asthma...) : Primary alveolar macrophages ( pAMs ) from naïve BALB/c mice were cultured to determine whether Mp-induced PGE ( 2 ) or exogenous PGE ( 2 ) down-regulates IL-4/IL-13 induced eotaxin-2 ... In pAMs, IL-4/IL-13 significantly increased eotaxin-2 , which was reduced by Mp infection accompanied by dose dependent PGE ( 2 ) induction ... Exogenous PGE ( 2 ) inhibited IL-4/IL-13 induced eotaxin-2 in a dose dependent manner