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CD79A — TNFSF13B

Text-mined interactions from Literome

Sakurai et al., Blood 2007 : Furthermore, TACI negatively regulated BAFF induced B-cell proliferation and production of IgA and IgG
Goto et al., Clin Immunol 2008 (Glomerulonephritis, IGA) : In this study, we focused on roles of BAFF and IFN-gamma in IgA production of TMCs stimulated with CpG-ODN in IgAN patients
Xu et al., J Immunol 2008 : Subsequent IgG and IgA production was enhanced by BAFF ( B cell activating factor of the TNF family ), an innate mediator released by TLR3 expressing mucosal dendritic cells
Kanswal et al., J Immunol 2008 : More importantly, TACI ligands BAFF or APRIL were unable to induce IgA/IgG/IgM secretion from newborn B lymphocytes
Crowley et al., Blood 2009 : These studies investigate how interactions between the BCR and FcgammaRIIB affect B lymphocyte stimulator (BLyS) recep-tor expression and signaling
Stohl et al., Arthritis Rheum 2010 (Lupus Erythematosus, Systemic) : In B6 and B6.Sle1 mice, constitutive BAFF overexpression led to increased numbers of B cells and CD4+ memory cells, as well as increased serum IgG and IgA autoantibody levels
Rowland et al., J Immunol 2010 : Additionally, we show that BAFF is essential for the differentiation of CD23 ( - ) into CD23 ( + ) transitional B cells both in vitro and in vivo through a mechanism distinct from that mediating cell survival, but requiring tonic BCR signaling
Tezuka et al., Immunity 2011 : IgA production was dependent on APRIL and BAFF production by pDCs
de Andrés et al., J Immunol 2012 : Moreover, in vitro stimulation of splenic 19?45R ( lo ) cells with LPS, CpG, BAFF/IL4 , and CD40/IL4 induced cell proliferation, IgG1/IgA secretion and the release of IL-10, suggesting a potential immunoregulatory role for this subset of innate-like B cells