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CASP14 — CXCL9
Text-mined interactions from Literome
You et al., Hepatology 2001
(Carcinoma, Hepatocellular...) :
Furthermore, apoptosis could be completely blocked by Z-VAD-FMK ( a general caspase inhibitor ) or by IETD-CHO ( a caspase-8 inhibitor ), but was only partially blocked by
Ac-DEVD-CMK ( a
caspase-3 inhibitor ), by N-acetyl-L-cysteine (NAC) ( an antioxidant ), by N-acetyl-leucyl-leucyl-norleucinal ( ALLN ) ( a calpain inhibitor I ), or by Z-LEHD-FMK ( a caspase-9 inhibitor )
Latorre et al., Eur J Biochem 2003
:
Furthermore, experiments with
Ac-DEVD-CMK (
caspase-3 specific
inhibitor ) have led us to conclude that caspase-3 is downstream of ceramide in activating the brain plasmalogen-selective phospholipase A2
Florio et al., J Cell Biochem 2003
(Leukemia, Lymphocytic, Chronic, B-Cell) :
Furthermore, both
caspase-3 activation and apoptosis triggered by IDA alone or in combination with MPA were significantly
inhibited by specific caspase-3 inhibitor
AC-DEVD-CMK
Chen et al., Mol Endocrinol 2013
(Diabetes Mellitus, Type 2) :
Using adenoviral vectors to overexpress or knockdown Mig6, we found that
caspase 3 activation during apoptosis was
dependent on the level of
Mig6
Iwasaki et al., Biochem Mol Biol Int 1997
(Carcinoma, Squamous Cell) :
Both
YVAD-CMK , an
inhibitor of
caspase-1 , and DEVD-CHO, an inhibitor of caspase-3, moderately inhibited the UVB induced cell death