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ATF1 — CAMK2A

Pathways - manually collected, often from reviews:

• OpenBEL Selventa BEL large corpus: ATF1 → CAMK2A (directlyIncreases, CAMK2A Activity, ATF1 Activity)
  Evidence: The substrate preferences of CaMKI and CaMKIV are similar and intersect with CaMKII. In vitro, all three can phosphorylate synapsin I, cAMP response element-binding protein (CREB), and activating transcription factor 1 (11, 12).

Protein-Protein interactions - manually collected from original source literature:

Studies that report less than 10 interactions are marked with *

• IRef Biogrid Interaction: ATF1 — CAMK2A (direct interaction, enzymatic study) Shimomura et al., J Biol Chem 1996*