We have a suspicion that you are an automated web bot software, not a real user.
To keep our site fast for other users, we have slowed down this page.
The slowdown will gradually disappear.
If you think this is a mistake, please contact us at genome-www@soe.ucsc.edu.
Also note that all data for hgGeneGraph can be obtained through our public MySQL server and
all our software source code is available and can be installed locally onto your own computer.
If you are unsure how to use these resources, do not hesitate to contact us.
Chakravarty et al., J Biol Chem 2001
:
The more than 10-fold induction of PKA stimulated PEPCK-C gene transcription caused by the co-activator CBP, was also blocked by SREBP-1c ... In addition, dnSREBP-1c reversed the strong negative effect of E1A and NF1 on PKA stimulated transcription from the PEPCK-C gene promoter
Banales et al., Hepatology 2009
(Cysts...) :
On the other hand, both PKA and Epac activation of cystic structures generated by normal and PCK cholangiocytes when cultured under three-dimensional conditions resulted in increased cyst growth, particularly in PCK-cholangiocyte derived cysts
Quinn et al., J Biol Chem 1994
(Liver Neoplasms, Experimental) :
The minimal promoter/transcription factor requirements for induction of phosphoenolpyruvate carboxykinase ( PEPCK ) transcription by cAMP activated protein kinase A (PKA) and inhibition of this induction by insulin were investigated