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CALM3 — KRAS

Pathways - manually collected, often from reviews:

• KEGG Long-term potentiation: CALM1/CALM2/CALM3/CALML3/CALML5/CALML6 → HRAS/KRAS/NRAS (protein-protein, activation)

Text-mined interactions from Literome

Villalonga et al., Mol Cell Biol 2001 : Furthermore, calmodulin inhibition preferentially activated K-Ras
Villalonga et al., J Biol Chem 2002 : In vitro experiments showed that the phosphorylation of K-Ras by PKC was inhibited by calmodulin , suggesting that calmodulin dependent modulation of K-Ras phosphorylation by PKC could be the mechanism underlying K-Ras activation in fibroblasts treated with TPA plus W13
Lopez-Alcalá et al., J Biol Chem 2008 : Furthermore, using a K-Ras with impaired binding to calmodulin but with membrane localization, we could demonstrate in striatal neurones that interaction between K-Ras and calmodulin was not required for Golgi K-Ras translocation induced by Ca ( 2+ ) influx