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ICAM1 — TGFB1

Text-mined interactions from Literome

Shimizu et al., Int J Lepr Other Mycobact Dis 1999 (Leprosy) : Roles of tumor necrosis factor-alpha and transforming growth factor-beta in regulating intercellular adhesion molecule-1 expression on murine peritoneal macrophages infected with M. leprae
Banu et al., Exp Nephrol 2002 : These studies demonstrate normal patterns of class II MHC, ICAM-1 , and B7 expression in TGF-beta1 ( -/- ) tubular epithelial cells in response to IFN-gamma, LPS, and TGF-beta1
Papon et al., Laryngoscope 2002 (Nasal Polyps) : Interferon-gamma significantly increased HLA-DR and ICAM-1 expression, whereas transforming growth factor-beta1 significantly decreased HLA-DR expression and lipopolysaccharide significantly increased ICAM-1 expression
Zhang et al., Am J Pathol 2004 (Lymphoma, Large B-Cell, Diffuse) : Removal of cell-surface HA increased ICAM-1 dependent monocyte binding and stimulation of TGF-beta1 synthesis
Hosokawa et al., Clin Exp Immunol 2006 : Transforming growth factor (TGF)-beta1 enhanced ICAM-1 expression
Hosokawa et al., Clin Exp Immunol 2006 (Periodontitis) : The ICAM-1 expression induced by TWEAK was augmented by TGF-beta1
Sawada et al., Adv Med Sci 2006 (Liver Neoplasms...) : By using three pancreatic cancer cell lines, SW1990, CAPAN-2 and PANC-1, the effect of TGF-beta1 on expression of ICAM-1 , cancer cell immunogenicity and liver metastasis were investigated
Phillips et al., Chang Gung Med J 2007 (Fibrosis) : In addition, direct interaction between monocyte-macrophage CD18 and PTC cell surface ICAM-1 stimulates TGF-beta1 synthesis
Krutmann et al., J Invest Dermatol 1990 : Stimulation of cells with recombinant human ( rh ) interleukin (IL) 1 alpha, rhIL-4, rhIL-5, rhIL-6, rh granulocyte/macrophage colony stimulating factor ( GM-CSF ), rh interferon alpha ( rhIFN alpha ), and rh transforming growth factor beta ( TGF beta ) did not increase ICAM-1 surface expression
Suzuki et al., J Appl Physiol 1994 : TGF-beta 1 causes increased endothelial ICAM-1 expression and lung injury ... We examined the effect of transforming growth factor-beta 1 ( TGF-beta 1 ) on the expression of ICAM-1 in human umbilical vein endothelial cells ( HUVEC ) ... TGF-beta 1 ( 1 ng/ml ) increased ICAM-1 and ICAM-1 mRNA expression in HUVEC, as assessed by flow cytometry and Northern blot analysis, respectively ... These results suggest that TGF-beta 1 causes neutrophil mediated lung injury, possibly through upregulation of ICAM-1 on endothelial cells, and might be important in the pathogenesis of lung injury
Xiao et al., Clin Exp Immunol 1996 : TGF-beta1 inhibited MBP stimulated and MBP activated glial cell proliferation, especially in MBP stimulated separated microglia and astrocytes, and down-regulated the expression of ICAM-1 on MBP stimulated glial cells and separated microglia ... Examination of TGF-beta1 and IFN-gamma interactions revealed that TGF-beta1 mediated inhibition of proliferation and down-regulation of ICAM-1 on glial cells were prevented by IFN-gamma
Paolieri et al., Allergy 1997 : IL-4, IL-6, IL-8, IL-10, and TGF-beta 1 did not affect either ICAM-1 expression or sICAM-1 release
Grünberg et al., Int J Mol Med 1998 : Pro alpha1 in combination with TGF-beta1 showed no significant effects on ELAM-1 expression, but antagonized the TGF-beta1 induced decrease of ICAM-1 and VCAM-1 expression on activated HUVECs