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FOS — TCF20

Text-mined interactions from Literome

Chen et al., Mol Carcinog 1999 : Our previous studies have indicated that UVB induced c-fos expression may play a key role in UVB induced activation of the activator protein-1 transcription factor and EGCG inhibited, UVB induced activation of AP-1 in HaCaT cells
Famulski et al., Cell Death Differ 1999 : We propose that ( i ) the up-regulation of an acidic nuclease activity is governed by a regulatory pathway different from that responsible for AP-1 factor induction, caspases activation and intracellular acidification, and ( ii ) activation of an acidic nuclease does not cause any deleterious effects when AP-1 transcription factor induction , caspases activation and intracellular acidification are down-regulated
Rutberg et al., Cancer Res 2000 (Carcinoma...) : A-FOS replaces endogenous Fos proteins in the formation of heterodimers with Jun family members and thus prevents the AP-1 transcription factor from binding to DNA
Troussard et al., Oncogene 2000 (Glioblastoma) : The integrin linked kinase (ILK) induces an invasive phenotype via AP-1 transcription factor dependent upregulation of matrix metalloproteinase 9 (MMP-9)
Bussolino et al., FASEB J 2001 : We show that, in addition to its transcription factor activity, c-Fos regulates the metabolism of phospholipids cytoplasmically by an AP-1 independent activity
Ventura et al., Mol Cell Biol 2003 (MAP Kinase Signaling System) : The JNK signal transduction pathway is therefore essential for AP-1 transcription factor regulation in cells exposed to TNF
Kinser et al., J Toxicol Environ Health 2004 (Inflammation) : c-Fos , Fra-, c-Jun, and JunB, components of the activator protein-1 (AP-1) transcription factor complex, were induced by DON as well as another transcription factor , NR4A1
Hwang et al., FEBS Lett 2005 : AP-1 activation suppressed the expression of Sox-9, a major transcription factor regulating type II collagen expression
Sahambi et al., Birth Defects Res A Clin Mol Teratol 2006 (Abnormalities, Drug-Induced) : We hypothesize that oxidative stress and activation of a redox-sensitive transcription factor , activator protein-1 (AP-1) , are early indicators of embryonic stress in response to a teratogenic insult
Tu et al., Am J Physiol Cell Physiol 2007 : Involvement of oxidants and AP-1 in angiotensin II-activated NFAT3 transcription factor
Weinberg et al., Neuroscience 2007 : Differential basal and stress induced expression patterns of the fra-2 and c-fos genes are likely to have important functional consequences for AP-1 transcription factor dependent regulation of neural plasticity
Lin et al., Inflamm Bowel Dis 2008 (Crohn Disease) : Probiotic L. reuteri suppressed TNF transcription by inhibiting activation of MAP kinase regulated c-Jun and the transcription factor , AP-1
Jung et al., Biofactors 2008 : Matrine inhibited PMA induced activation of the AP-1 promoter, an important nuclear transcription factor in MMP-1 expression
Yen et al., J Biol Chem 2011 (MAP Kinase Signaling System) : PKA, PI3K, and ERK inhibitors abolished PGE2- and cAMP induced c-Fos and MMP-9 up-regulation, and ERK activation was required for the binding of activator protein 1 (AP-1) transcription factor to the MMP-9 promoter
Chae et al., Biosci Biotechnol Biochem 2011 : And, it inhibited DNA binding activity of activator protein-1 (AP-1) , a transcription factor of MMP-1 and downstream of ERK and JNK
Hattori et al., Am J Physiol 1993 (Acute-Phase Reaction...) : These findings demonstrate the complexity of AP-1 hepatic transcription factor responses to humoral regulators with direct hepatocellular effects
Erkel et al., J Antibiot (Tokyo) 1996 : In reporter gene assays nidulal ( 1 ) preferentially activated the transcription factor complex AP-1 mediated expression of secreted alkaline phosphatase in COS-7 cells
Manna et al., J Immunol 1998 : TNF induced activation of another nuclear transcription factor , AP-1 , was suppressed by IL-13
Chen et al., J Biol Chem 1998 : The effects of UVB irradiation on transcription factor activator protein-1 (AP-1) DNA binding and AP-1 transactivation were studied in a human keratinocyte cell line, HaCaT