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CSF2 — ICAM1

Text-mined interactions from Literome

Wanner et al., Langenbecks Arch Surg 1999 (Endotoxemia) : rG-CSF per se did not affect NOS II, CINC, or ICAM-1 expression in hepatic liver cells, while LPS induced a marked expression of NOS II, CINC, and ICAM-1 in nonparenchymal cells and, to a lesser extent, in hepatocytes
Wong et al., Am J Respir Cell Mol Biol 2003 : IL-3, IL-5, and GM-CSF could enhance p38 MAPK and NF-kappaB activity and induce ICAM-1 , CD11b, and CD18 expressions on eosinophils
Detmar et al., J Invest Dermatol 1992 : The effects of recombinant human interleukin 1 alpha (IL-1 alpha), interleukin 1 beta (IL-1 beta), interleukin 6 (IL-6), granulocyte/macrophage colony stimulating factor ( GM-CSF ) and tumor necrosis factor-alpha (TNF) on the cell proliferation and the expression of intercellular adhesion molecule-1 ( ICAM-1 ) were assessed in cultured human dermal microvascular endothelial cells ( HDMEC )
Lu et al., Invest Ophthalmol Vis Sci 2006 : Lipopolysaccharide (LPS) induced the release of the proinflammatory cytokine IL-6 and that of the chemokines G-CSF, MCP-1, MIP-1beta, and IL-8 as well as surface expression of ICAM-1 by corneal fibroblasts, whereas IL-1beta, TNF-alpha, and GM-CSF were not detected in the culture supernatants of cells incubated with or without LPS
Krutmann et al., J Invest Dermatol 1990 : Stimulation of cells with recombinant human ( rh ) interleukin (IL) 1 alpha, rhIL-4, rhIL-5, rhIL-6, rh granulocyte/macrophage colony stimulating factor ( GM-CSF ), rh interferon alpha ( rhIFN alpha ), and rh transforming growth factor beta ( TGF beta ) did not increase ICAM-1 surface expression
Bernasconi et al., Int J Cancer 1995 (Adenocarcinoma...) : The protein synthesis inhibitor cycloheximide super induced basal and GM-CSF induced ICAM-I transcripts, thus excluding a role for secondary polypeptide mediators
Czech et al., J Invest Dermatol 1993 (Hypersensitivity, Immediate) : The combination of TNF alpha with either IFN gamma, IL-3, IL-5, or granulocyte/macrophage colony stimulating factor ( GM-CSF ) increased ICAM-1 expression in a synergistic fashion, whereas IL-5 or GM-CSF by itself did not induce ICAM-1 expression ... In addition, these data demonstrate for the first time a role for IL-3, IL-5, and GM-CSF in regulation of ICAM-1 expression in human cells
Savage et al., J Pathol 1997 : Glomerular endothelial ICAM-1 expression was increased by IL-1, TNF, IFN-gamma, and GM-CSF , while TGF beta inhibited induction by TNF and IL-1
Horie et al., Immunology 1997 : Recombinant GM-CSF alone induced a slight but significant CD54 expression on eosinophils
Nagata et al., Am J Respir Cell Mol Biol 1998 (Asthma...) : Granulocyte macrophage colony stimulating factor augments ICAM-1 and VCAM-1 activation of eosinophil function